The so-called climate crisis is a piss in the ocean compared to the Testosterone-crisis humanity is now facing. I hope you have at least somewhat oriented yourself about this. Even contemporary scientists are now starting to address the problem, although they hide it behind “Sperm-count”- which is in pretty much every case completely proportional with “Free T-count”. Just the last thirty days there’s been two quite well written articles on the subject even in one of Norway’s top two Mainstream Newspapers- I think this is very good. One of these articles you can read right her (in English).
As I have written in several articles before(I advise you to read them all, they are available right here on the website), the T-levels in men have dropped an alarming 50% (or more) during the last 50 years. The decline is by no means proportional- so it’s not 1% a year- more probably it was around 0,2% during the first decade from 1970-1980, and then it just increased every year, until now it probably drops more than 2% every year. You don’t need to be specifically good a maths to understand that within a few decades(or actually right now) men are no longer Men- except those who realize the problem and actually make an effort to fight against it. The sperm-count have dropped 52% during the same time span- so as you can see it’s completely proportional and parallel. Even at this time there are countless men who can no longer re-produce, simply because they ain’t men enough. It’s a sad story, isn’t it.
Let’s take a closer look at all the comparisons above:
Testosterone and Libido
Along with the muscle building factors, this is probably the one factor that at least most men are aware of when it comes to how Testosterone works in the body. Low T equals low sex drive- High T equals higher sex drive and capability. Testosterone is the “gasoline” of our sexual performance, of our sex organs literally- speaking for the men now. If you ever experienced ED (erectile dysfunctioning) this is highly probably due to a lack of free T in the bloodstream. Resorting to shortcuts like Viagra will only make the matter worse. You gotta identify the source, which lies in low T, not artificially just adjust certain processes like Viagra does. Using such substances for a long time can and will eventually lead to chronic ED. Neither should you resort to TRT(testosterone replacement therapy), as this will furthermore shut down your body’s natural production. There are numerous excellent remedies for this found in nature, both as nurtritive remedies, i.e herbs etc, and as protocols you can follow. I strongly advise you to read this article ‘Testosterone- The Elixir of Life’ for detailed information on what you can do to NATURALLY optimize your own T.- It’s available for free as an e-book for customers in my store.
If you want an idea on which natural remedies you can take to optimize your natural production of Testosterone; do take a closer look at these:
And for easier “All in One Solutions”, take a look at these two formulas:
Testosterone and Muscle Mass vs Fat
This is of course what most people connect with testosterone; muscles- and there’s a very good reason for it. Testosterone is the absolute no.1 factor when it comes to building strong, lean muscle mass. It’s also on the opposite end, when there’s lack of it, the number one factor contributing to overweight and increased BMI. Low T leads almost without exception to the accumulation of fatty tissue, especially around the waist area of men; this in turn increases Aromatase, which is an enzyme that transforms Testosterone into Estrogen, making the matter worse. This can become a viscious cycle; low T accumulates waist fat; waist fat in turn accumulates aromatase, which further “kills” T by transforming it into Estrogen. This makes it very hard to get rid of the excessive fat again, knowing that Testosterone is the No.1 fat burner. Be extremely vary of waist fat; as it will, completely without exception lessen your T-levels.
Higher natural level of Testosterone is what makes men in general considerably stronger than women physically, and that’s an undisputable fact. That being said; Testosterone is, just the same as in men, also the main factor contributing to muscle growth in women.
Mental faculties
It has been proven without doubt that there is a clear connection between testosterone and one’s mental faculties. The higher the level of free T flowing in your blood, and penetrating the blood-brain barrier, the sharper your mind and faculties will be- especially so on the manly traits of for example spatial recognition. Yes, men excel at this, that has also been proven beyond doubt. Women excel at a lot of things, but that is not the theme of this article.
“Hormone administration studies have also provided useful insights into the factors associated with variations in cognitive performance, particularly in relation to the established gender differences that exist in cognitive functioning. It is well known that on average, men generally outperform women on visuospatial tasks and women outperform men on verbal fluency and perceptual speed tasks (Halpern, 2000). Of course, it is important to note that the overlap in performance between men and women on each of these tasks is much greater than the mean differences between the sexes. Nonetheless, several biologically plausible mechanisms have been proposed to account for these differences. Evidence suggests that endogenous sex hormones affect cognitive functioning through their
pre- and perinatal effects on sexually dimorphic brain structures (Collaer & Hines, 1995). For example, in a seminal study Hier and Crowley (1982) showed that androgen deficient (hypogonadal) men exhibited a marked deficit in visuospatial ability compared with matched controls and hypogonadal men who acquired the condition post-pubertally. These findings indicate that testosterone has an organisational effect on the normal expression of spatial ability. More recently, we found testosterone to also have activational effects on cognitive functioning in healthy men: improvements in verbal ability were found to accompany a reduction in spatial ability following testosterone treatment (O’Connor et al., 2001a). These findings suggest that the relationship between testosterone and cognitive functioning is not straightforward, and that an optimum level of hormone is required for the normal expression of spatial ability.
Age-related reductions in testosterone have been found to be associated with a progressive decline in cognitive abilities. In fact, several studies have examined whether testosterone supplementation in older men can benefit cognition. Exciting recent developments in this area have shown that hormone replacement therapy in men can have beneficial effects on aspects of cognitive functioning. Cherrier and colleagues (2007) found that moderate-to-high increases in testosterone were associated with significant improvements in verbal and spatial memory. This could be a promising avenue for future research, with implications for treating older men with existing cognitive difficulties such as mild cognitive impairment or Alzheimer’s disease.”(1)
– The British Psychological Society
Testosterone and heart health
There is a very clear connection between your level of free testosterone and the healthiness of your heart, this has been proven by numerous studies done on the subject. Lower testosterone levels almost without exception leads to lower level of physical activity- which again often leads to weight gain and a number of health problems, including decline in artery health and function. This is just one side of it, the whole thing is really quite complex:
Testosterone actions on cardiovascular cells
Testosterone and the more potent DHT bind to cytoplasmic androgen receptors (AR) that are chaperoned by heat shock proteins. Once bound, the DHT-AR complex migrates to the nucleus, dimerizes with another DHT-AR complex, associates with coactivator proteins, and transactivates a family of genes with androgen response elements that alter myocardial and vascular cell behavior.1 Evidence also supports a direct rapid membrane effect of T on G-protein-coupled receptors, with an increase in inositol trisphosphate and diacylglycerol and subsequent alterations in cytoplasmic calcium and potassium channel activity.(1)
Cardiovascular Health in Testosterone Deficiency
Testosterone deficiency (TD) is a well-established major medical condition that negatively impacts male sexuality, general health, and quality of life. Symptoms include decreased libido, erectile dysfunction, decreased energy, depressed mood, irritability, and decreased sense of well-being. In the correct clinical setting, the diagnosis of TD is usually confirmed by low serum concentrations of total T (e.g., < 200 ng/mL) drawn in the early morning. However, there is no specific value that reliably distinguishes men who experience signs and symptoms of TD from those who do not nor those who will likely respond to treatment. Interpretation of total T concentrations is confounded by variation between individuals, variation in serum SHBG, and variation in androgen sensitivity.6 Furthermore, considerable controversy has arisen regarding the accuracy of currently available commercial testosterone assays, especially those showing T levels at the lower end of the “normal” range.4 Free testosterone level may be a more reliable indicator of androgen status, but more studies are needed to confirm this. The prevalence of symptomatic TD ranges from 2.1% to 12.8% in middle-aged to older men, with an incidence of 12 new cases/1000 person-years in the United States and Europe. Populations at high risk for TD include men with CHF, type 2 diabetes, obesity, chronic obstructive pulmonary disorder, HIV, and chronic opioid use.(1)
Overall and Cardiovascular Mortality in Testosterone Deficiency
Low T levels are also associated with chronic medical conditions such as metabolic syndrome, diabetes, dyslipidemia, hypertension, renal failure, frailty, malignancy, and cardiovascular (CV) events. Several meta-analyses and systematic reviews have clearly associated TD with increased CV disease and mortality. Ruige et al. found that higher T levels were associated with a decreased risk for CV events in men > 70 years (HR of 0.84; 95% CI, 0.76–0.92) but not in younger men (HR of 1.01; 95% CI, 0.95–1.08).8 In a meta-analysis by Araujo et al. that included 18 studies and more than 22,000 subjects, overall and CV mortality were related to T levels. The authors concluded that although there was considerable heterogeneity in the studies, low T levels were significantly associated with overall mortality and strongly (P = .06) associated with CV mortality. 9 Finally, Corona et al. screened 1,178 articles and found 70 in their meta-analysis that showed a clear association between low T/high estradiol levels and CV disease.10 Longitudinal studies demonstrated that overall mortality and CV mortality were highest in those with low T levels. Whether low T and increased mortality are simply covariates or a causal relationship remains to be proven.(1)
Testosterone Deficiency and Coronary Artery Disease
In their 2013 review, Oskui and colleagues reported on evidence suggesting that men with lower levels of endogenous T are more likely to develop CAD during their lifetimes.11 The severity of CAD has also been investigated as a function of serum T concentrations. Four studies have noted an inverse relationship between serum T levels and CAD severity,6 with lower levels of serum T associated with more severe CAD and higher serum T levels associated with reduced severity.11 However, these results should be interpreted with caution due to the relatively small sample size included in each study. Furthermore, the mechanism through which TD may exacerbate CAD is unknown. Additional research is needed to further evaluate the association between low T levels and CAD severity. Testosterone may have several types of action, including direct vasodilatory action, a direct effect on myocardial oxygen consumption, and membrane repolarization.5,11
Testosterone Deficiency and Congestive Heart Failure
Emerging evidence indicates that congestive heart failure (CHF) is more than just a syndrome affecting a failing heart. It is becoming clear that the pathophysiology of CHF involves other pathways as well, including the skeletal muscles and the endocrine system. Jankowska et al. studied 208 men with CHF and 366 healthy male controls. Low T levels were found in all NYHA classes of heart failure.12 It has also been shown that reduced T levels in men with CHF portends a poor prognosis and is associated with increased mortality.13
Testosterone and Bone Density
It has also been proven beyond any doubt how Testosterone impacts the healthy status of our bone structure. High T means stronger, denser bones; while low T consequently will lead to more brittle bones and Osteoporosis.
Figure explanation: Sex hormones and the bone, in men. Testosterone is secreted from the testes and bound to sex hormone–binding globulin (SHBG) or albumen. SHBG is made mainly in the liver and is influenced by many factors, including the sex hormones themselves. Aromatase converts some of the free testosterone to estrogen (which also can bind to the SHBG). The free hormones then activate receptors on the bone cells. It is unclear whether SHBG has a receptor on the bone cells. Estrogen acts mainly on the cortex with some effects on the trabecular bone, and testosterone acts on the trabecular bone. Testosterone also increases periosteal expansion. CKD, chronic kidney disease; LH, luteinizing hormone.
Sex hormones play a major role in the growth and maintenance of the skeletal system. Androgens mediate the periosteal growth in both genders. Because men have higher androgen levels they have greater cortical thickness and peak bone mass. The periosteum continues to expand throughout life, more so in men. Because cortical bone is placed further away from the neutral axis, there is greater resistance to bending. This helps to compensate for aging bone loss.1
In the past it was assumed that estrogen regulated bone metabolism in women, whereas testosterone performed these functions in men. An interventional study in normal men, all treated with gonadotropin blockade and aromatase inhibition and then randomized into 4 groups (receiving placebo, estrogen, testosterone, or both), showed that estrogen accounted for more than 70% of the effect of sex steroids on bone resorption. Since then, numerous studies in men have shown that bioavailable estrogen is more closely associated with bone density than bioavailable testosterone. But the site-specific actions of sex hormones are different in men than in women. Estrogen maintains cortical bone (which is 80% of the bone mass) in both men and women. In women, estrogen is the main hormone that maintains the cancellous bone, with small additional actions from testosterone, whereas in men the testosterone is the major hormone in the cancellous bone with minor contributions from estrogen(1)
Strength, Courage, Mastery, and Honor are the alpha virtues of men all over the world
Jack Donovan
Norsk bokmål 
English (UK)